Remergent Notes:

DNA Science for Skin Platform

Developed by Dr. Daniel B. Yarosh, a molecular biologist with a specialty in gene repair, Remergent is backed by better than twenty years of research and clinical study. Dr. Yarosh, who gained experience as an expert in DNA repair at the National Cancer Institute in Bethesda, Maryland, pioneered the development of a unique molecular delivery system. The patented technology is based on inserting repair enzymes into liposomes (nanometer lipid sacs) which are small, pure and agile enough to deliver payload ingredients at the cellular level.

Validated in controlled clinical studies and published in dozens of peer-reviewed scientific and medical journals, liposome-delivery is at the core of Remergent technology.

Damaging Effects of Sun on Skin

Exposure to ultraviolet light from sunlight accounts for nearly all of the symptoms of premature skin aging. Other skin changes, once believed to be caused by natural aging, are now known to be a result of prolonged exposure to UV radiation, called photoaging.

The ultraviolet radiation from the sun that reaches the earth’s surface—and your skin—is divided into two categories: UVA and UVB, based on their wavelengths. The more energetic UVB rays are the primary agents for sunburn and do most of their damage in the uppermost layers of living skin. They have been closely linked to skin cancer. The far more abundant UVA rays have less energy but penetrate deeper into skin, exposing the underlying cells and supporting collagen fibers.

Both UVA and UVB cause damage to the skin’s DNA, the master control for all the cell’s functions. Damaged cells send out stress signals to other cells, and this begins a cascade of wounding responses that leads to premature aging. If the DNA damage is not repaired properly, the skin cell loses its ability to function correctly. Damaged cells may begin to divide and grow out of control, causing skin cancer.

The cascade of wounding responses has many effects on skin cells, structures and systems:

• Cells release enzymes that break down collagen at a faster rate, causing collagen fibers to fray. They separate from living cells, which makes the healing process more difficult. The result is a disorganized network of collagen fibers, known as solar scars. When the skin repeats this imperfect rebuilding process over years of sun exposure, wrinkles develop.
• Melanocytes are stimulated to make pigment (melanin) in the skin. In youth, for some, the result may be a tan. But during photoaging, the melanocytes lose their strict controls, and uneven pigmentation develops. This appears as light spots where melanocytes have stopped working or as dark brown areas, called age or sun spots, where they make too much melanin. 
• Blood vessels the in the skin’s surface permanently dilate and their walls thin. They appear as broken capillaries and spider veins (telangectasia). 
• The upper layer of skin fills up with flaky dead layers that do not hold moisture. The skin appears sallow and dry, exaggerating the appearance of lines and wrinkles. 
• Cells of the skin’s immune system are suppressed, severely reducing their ability to eliminate damage or protect the skin from invading cells and toxins.